William Halsted
William Halsted (1852–1922), pioneering American surgeon, revolutionized surgery with aseptic technique, anesthesia, gloves, and the residency training model.
Category Medical Specialty
Adult Ortho Cases 005
Xray and ultrasound (POCUS) evaluation of integrity of quadriceps tendon, patella tendon, and patella evaluating for tendon rupture and patella fracture.
Meigs syndrome
Meigs syndrome: Triad of ascites with hydrothorax in association with benign ovarian tumor, that is cured after tumor resection. Described in 1934 by Joe Vincent Meigs (1892-1963)
Joe Vincent Meigs
Joe Vincent Meigs (1892-1963) American gynaecologic oncology. Meigs syndrome, radical hysterectomy innovations and validation of Pap smear screening.
Friedrich Horner
Swiss ophthalmologist Johann Friedrich Horner (1831–1886), eponym of Horner's syndrome, advanced ophthalmic surgery and neuroanatomical diagnostics
Chilaiditi syndrome
Chilaiditi sign: rare condition with bowel loops interposed between liver and diaphragm, with symptoms (syndrome). Must differentiate from free air.
Horner Syndrome
Horner syndrome is associated with an interruption to the sympathetic nerve supply of the eye. It is characterized by the classic triad of miosis, partial ptosis, and anhidrosis +/- enophthalmos
AF Stanley Kent
A. F. Stanley Kent (1863–1958), cardiac physiologist; 'bundle of Kent', shaped early electrophysiology; pioneered industrial fatigue science
Frieda Robscheit-Robbins
Frieda Robscheit-Robbins (1888–1973), pioneering pathologist who advanced aneamia research, yet was overlooked for the 1934 Nobel despite major contributions.
Bartter Syndrome
Bartter Syndrome: Renal tubulopathy with hypokalaemic alkalosis, high renin and aldosterone, normal BP, and polyuria—mimics loop diuretic effect at the thick ascending limb.
William B. Schwartz
William B. Schwartz (1922–2009) American nephrologist. Co-described SIADH, pioneered decision theory, medical AI, and ethical health care rationing.
Schwartz–Bartter syndrome
Schwartz-Bartter Syndrome (SIADH): Excess ADH leads to water retention and dilutional hyponatraemia, with low serum sodium and osmolality but inappropriately concentrated urine.