Polyuria and Traumatic Brain Injury
polyuria following TBI is common; multiple causes, some of which imply a poor prognosis. Goal is to identify and treat the cause and any complications
Moderate Head Injury
DEFINITION PROGNOSTIC DETERMINANTS FACTORS WARRANTING ADMISSION TO ICU Patient factors Environmental factors References and Links
Diabetes Insipidus following TBI
Diabetes insipidus (DI) results from decreased secretion and action of anti-diuretic hormone (ADH). ADH is produced in the hypothalamus and transported to the posterior pituitary gland
Brain Impact Apnoea
Brain impact apneoa is an under-appreciated cause of morbidity and mortality in traumatic brain injury (TBI)
Minimum Inhibitory Concentration
Minimum Inhibitory Concentration (MIC) is lowest concentration of an antimicrobial that will inhibit the visible growth of a microorganism following overnight incubation, usually reported as mg/L
Rectal mass
Rectal masses may be intra-luminal, mural or palpable through the rectum but arising from outside of it. Differential diagnosis.
Splenomegaly DDx
Splenomegaly refers to enlargement of the spleen. Differential diagnosis
Limitations of CT in Traumatic Brain Injury
Limitations of CT head scan in Traumatic Brain Injury (TBI)
Antibiotic Dosing in Renal Replacement Therapy
Little published data to guide dosing of antibiotics in RRT and the critically ill. therefore we must use first principles and utilize therapeutic drug monitoring if available.
Laboratory Urinalysis
Laboratory urine analysis
Cerebral Perfusion Pressure in TBI
Cerebral Perfusion Pressure (CPP) in Traumatic brain injury (TBI). Cerebral Perfusion Pressure (CPP) = MAP – ICP or CVP (whichever is highest)
Bronchiolitis
Goals: resuscitation severity and stability of disease assessment of possible differentials including cardiac disease, influenzae, pertussis stabilisation and possible transfer to neonatal/SCBU if required