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NSAIDs

Nonsteroidal anti-inflammatory drugs (NSAIDs)

CLASS

  • non-selective COX inhibitors

Types – weak organic acids

  1. Salicylic acids – aspirin
  2. Acetic acids – diclofenac, ketorolac, indomethacin
  3. Propionic acids – ibuprofen, naproxen
  4. Fenamates – mefenamic acid
  5. Oxicams – tenoxicam, piroxicam
  6. Paracetamol
  7. Pyrazolones – phenylbutazone

MECHANISM OF ACTION

  • cyclooxygenase (COX) is an enzyme that catalyses the synthesis of prostaglandins from arachidonic acid.
  • prostaglandins mediate a large number of body processes including:
    1. inflammation
    2. pain
    3. secretion of a protective gastri layer
    4. maintenance of renal perfusion
    5. platelet aggregation
  • NSAIDs block the action of COX -> reducing production -> results:
    1. analgesia
    2. anti-inflammation
    3. decreased gastric mucosa -> ulceration
    4. decreased renal perfusion
    5. bleeding

Main actions

  • inhibition of biosynthesis of prostaglandins
  • also:
    1. inhibition of chemotaxis
    2. down regulation of interleukin-1 production
    3. interference with Ca2+ mediated intracellular events
    4. decrease sensitivity of vessels to bradykinin & histamine
    5. affect lymphokine production from T lymphocytes
    6. reverse vasodilation
    7. inhibit platelet aggregation

ADVERSE EFFECTS

  • bronchospasm
  • hyperventilation
  • GI ulceration, haemorrhage
  • hepatic dysfunction -> transaminitis
  • renal dysfunction from excretion of hypertonic metabolites -> papillary necrosis
  • inhibition of platelet aggregation
  • prolongation of bleeding time
  • hyperglycaemia -> glycosuria

PHARMACOKINETICS

  • Absorption – most are well absorbed, absorption not affected by food
  • Distribution – highly protein bound (>98%), all are found in joints after repeated dosing
  • Metabolism – hepatic
  • Elimination – renal most important, biliary -> enterohepatic circulation

EVIDENCE

-> effective analgesics of similar efficacy for acute pain
-> NSAIDs + paracetamol = more analgesia
-> with careful selection of patients NSAID induced renal impairment is low
-> aspirin + some NSAIDs increase perioperative bleeding after tonsillectomy except in paediatrics
-> NSAIDs reduce opioid consumption
-> NSAIDs increase perioperative blood loss

CCC Pharmacology Series

Critical Care

Compendium

Chris is an Intensivist and ECMO specialist at The Alfred ICU, where he is Deputy Director (Education). He is a Clinical Adjunct Associate Professor at Monash University, the Lead for the  Clinician Educator Incubator programme, and a CICM First Part Examiner.

He is an internationally recognised Clinician Educator with a passion for helping clinicians learn and for improving the clinical performance of individuals and collectives. He was one of the founders of the FOAM movement (Free Open-Access Medical education) has been recognised for his contributions to education with awards from ANZICS, ANZAHPE, and ACEM.

His one great achievement is being the father of three amazing children.

On Bluesky, he is @precordialthump.bsky.social and on the site that Elon has screwed up, he is @precordialthump.

| INTENSIVE | RAGE | Resuscitology | SMACC

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